Audrey Bedouch Ostéopathe

18/05/2026

L’endométriose est une maladie féminine qui se propage à l’extérieur de l’utérus.
L’ostéopathie peut aider à diminuer les douleurs liées à cette maladie en relâchant les muscles et en restaurant la mobilité des pièces osseuses entraînant par la même occasion une activation de la circulation sanguine et donc libérer les facteurs inflammatoires.
N’hésitez pas à consulter votre ostéopathe.
Audrey Bedouch Ostéopathe

🎗️ Endometriosis & Neuropathic Pain: The Lateral Femoral Cutaneous Nerve Connection
When we think about endometriosis, deep pelvic pain, severe cramping, and GI distress are usually the first symptoms that come to mind. But what happens when endometriosis causes burning, tingling, or numbness down the outside of your thigh?
It might be Meralgia Paresthetica triggered by extra-pelvic or deep infiltrating endometriosis.
🔍 The Anatomy of the Compression
The Lateral Femoral Cutaneous Nerve (LFCN) arises from the L2-L3 nerve roots, courses through the pelvis, runs along the pelvic sidewall under the iliac fascia, and exits into the thigh beneath the inguinal ligament.
Because of this specific anatomical pathway, the LFCN is vulnerable to irritation from endometriosis in a few key ways:
Direct Lesions: Endometriosis implants or chocolate cysts residing on the pelvic sidewall can directly infiltrate or compress the nerve sheath.
Fibrosis & Scarring: Cyclic bleeding from lesions causes localized inflammation, leading to dense adhesions and scar tissue that trap the nerve against rigid structures like the inguinal ligament.
Inflammatory Cascades: Even without direct physical entrapment, the biochemical storm (prostaglandins and cytokines) from nearby pelvic lesions can irritate the nerve fibers, leading to neuropathic symptoms.
⚡ Recognizing the Symptoms
Unlike sciatica or femoral nerve pathology, the LFCN is a purely sensory nerve. This means its compression presents with distinct characteristics:
🔥 Sensation: Burning, tingling ("pins and needles"), hypersensitivity to clothing touch, or complete numbness.
📍 Distribution: Confined strictly to the anterolateral (outer-front) and lateral (outer) aspect of the thigh.
🚫 No Motor Weakness: Because it carries no motor fibers, it does not cause true muscle weakness or foot drop. However, severe pain can sometimes cause a protective limp or altered gait.
🛠️ The Clinical Approach
Managing nerve-related endometriosis requires a multidisciplinary strategy:
Targeted Physical Therapy: Neurodynamics (nerve gliding), myofascial release of the iliopsoas and tensor fasciae latae, and pelvic floor rehab to reduce surrounding muscular tension and improve nerve mobility.
Medical & Surgical Intervention: Specialized laparoscopic excision surgery to remove deep infiltrating lesions along the pelvic sidewall, paired with targeted pain management (like nerve blocks or neuromodulators) if neuropathy persists.
Awareness is key. If you or your patients are experiencing unexplained outer thigh burning alongside a history of pelvic pain, it's time to look closely at the lateral pelvic anatomy.

17/05/2026

Votre ostéopathe, votre meilleur allié pour les douleurs et maux de tête provenant de vos cervicales!! N’hésitez pas à consulter!!
Audrey Bedouch Ostéopathe

📚 Céphalées cervicogènes : que dit la science sur les thérapies manuelles ?

https://pubmed.ncbi.nlm.nih.gov/36419164/

Une récente r***e systématique et méta-analyse portant sur 20 essais cliniques randomisés (1439 patients) s’est intéressée à l’efficacité des thérapies manuelles et des exercices dans la prise en charge des céphalées cervicogènes.

🔎 Résultats principaux :
Les données suggèrent que les thérapies manuelles et les exercices peuvent permettre :
✔️ une diminution de l’intensité des douleurs
✔️ une réduction de la fréquence des céphalées
✔️ une amélioration du handicap fonctionnel
➡️ à court comme à long terme.

Les interventions étudiées comprenaient notamment :
• les manipulations vertébrales,
• les mobilisations cervicales,
• le traitement des trigger points,
• les exercices cranio-cervicaux et scapulo-thoraciques.

📈 Lorsque seules les études méthodologiquement les plus solides étaient analysées, les résultats restaient favorables, en particulier pour les manipulations vertébrales, avec un niveau de preuve jugé modéré.

🧠 En résumé : les données scientifiques actuelles soutiennent l’intérêt des approches manuelles et de l’exercice dans les céphalées cervicogènes, avec des résultats encourageants sur la douleur et la qualité de vie.

28/04/2026

Vous avez des douleurs en étaux autour du bassin et se diffusant vers l’avant?
Cela peut venir des nerfs des 1ere lombaires, appelée syndrome de Maigne.
Si vous ressentez des douleurs ou des modifications de sensibilité au niveau du bassin, n’hésitez pas à venir consulter votre ostéopathe pour débloquer la zone.
Audrey Bedouch Ostéopathe

𝐌𝐚𝐢𝐠𝐧𝐞 𝐒𝐲𝐧𝐝𝐫𝐨𝐦𝐞: 𝐓𝐡𝐞 𝐎𝐯𝐞𝐫𝐥𝐨𝐨𝐤𝐞𝐝 𝐒𝐨𝐮𝐫𝐜𝐞 𝐨𝐟 𝐋𝐨𝐰 𝐁𝐚𝐜𝐤, 𝐇𝐢𝐩, 𝐚𝐧𝐝 𝐆𝐫𝐨𝐢𝐧 𝐏𝐚𝐢𝐧

Low back pain is one of the most common musculoskeletal complaints worldwide. Yet in some patients, the true cause of pain is frequently overlooked: Maigne Syndrome, also known as thoracolumbar junction syndrome.

Recent literature continues to highlight that this condition is underdiagnosed but highly treatable when properly recognized.

👉 What Is Maigne Syndrome?

Maigne Syndrome refers to pain originating from dysfunction at the thoracolumbar junction, typically around the T12–L1 spinal level.

This region represents a biomechanical transition zone between the relatively rigid thoracic spine and the more mobile lumbar spine, making it particularly vulnerable to mechanical stress and dysfunction.

The syndrome was first described by French physician Dr. Robert Maigne in the 1980s.

👉 Pathophysiology

Recent reviews describe two main mechanisms:

1️⃣ Central (Facet Joint) Variant

Pain originates from facet joint dysfunction or arthropathy at the thoracolumbar junction.

2️⃣ Peripheral (Nerve Entrapment) Variant

Pain occurs due to entrapment of the superior cluneal nerve, which arises from the posterior rami of the lower thoracic and upper lumbar nerves.

Both mechanisms can generate referred pain patterns far from the spine, which explains why the syndrome is frequently misdiagnosed.

👉 Typical Pain Distribution

One of the reasons Maigne Syndrome is often missed is that pain may not be located in the spine itself.

Patients may present with pain in:

• Iliac crest
• Posterior pelvis
• Lateral hip
• Groin
• Lower abdomen
• Pseudo-sciatica symptoms

These referred pain patterns result from irritation of the thoracolumbar dorsal rami and related nerves.

👉 Key Clinical Signs

Several clinical findings can suggest Maigne Syndrome:

✔ Tenderness at the thoracolumbar junction (T12–L1)
✔ Pain reproduced by palpation of the posterior iliac crest
✔ Positive pinch-roll (skin rolling) test
✔ Localized hyperalgesia over the iliac crest

Diagnostic confirmation may involve local anesthetic nerve blocks, which can temporarily eliminate symptoms if the diagnosis is correct.

👉 Why It Is Frequently Misdiagnosed

Maigne Syndrome often mimics other conditions such as:

• Lumbar disc pathology
• Sacroiliac joint dysfunction
• Hip disorders
• Inguinal or abdominal pathology

Because of its atypical pain referral patterns, many patients undergo extensive imaging or treatments without identifying the true source of pain.

👉 Evidence-Based Treatment Approaches

When recognized correctly, treatment outcomes are generally favorable.

Common approaches include:

Conservative management

• Manual therapy targeting the thoracolumbar junction
• Spinal mobilization or manipulation
• Exercise therapy and stabilization programs
• Anti-inflammatory medications

Interventional options

• Diagnostic and therapeutic nerve blocks
• Cluneal nerve injections
• Surgical decompression in refractory cases

Patients frequently respond well to manual techniques combined with targeted exercises.

📌 Clinical Takeaway

Maigne Syndrome remains a hidden contributor to low back and pelvic pain.

For clinicians working with musculoskeletal pain, considering the thoracolumbar junction as a potential pain generator may prevent misdiagnosis and lead to more effective treatment.

Sometimes the pain is not coming from the lumbar spine itself—but from the small transition zone just above it.

✅ References (Recent Literature)
• Randhawa et al., 2022 – Maigne Syndrome: A potentially treatable yet underdiagnosed cause of low back pain
• Tsur & Ohry, 2024 – Thoracolumbar Junction Syndrome
• Singh & Kumar, 2022 – Pelvic pain in Maigne’s syndrome: a multi-segmental approach

13/04/2026

Un très bel article documenté sur le lien entre les douleurs de la région lombaire et le système du microbiote intestinal.

Les douleurs chroniques lombaires peuvent être alimentées par un intestin poreux laissant passer « les mauvaises bactéries », celles-ci migrant par le système sanguin vers les structures de la colonne lombaire.

La santé du dos passe aussi par la santé de l’intestin!

𝗣𝗮𝘁𝗶𝗲𝗻𝘁𝘀 𝗪𝗶𝘁𝗵 𝗖𝗵𝗿𝗼𝗻𝗶𝗰 𝗟𝗼𝘄 𝗕𝗮𝗰𝗸 𝗣𝗮𝗶𝗻 𝗪𝗶𝘁𝗵𝗼𝘂𝘁 𝗔𝗱𝘃𝗮𝗻𝗰𝗲𝗱 𝗗𝗶𝘀𝗸 𝗗𝗲𝗴𝗲𝗻𝗲𝗿𝗮𝘁𝗶𝗼𝗻 𝗘𝘅𝗵𝗶𝗯𝗶𝘁 𝗚𝘂𝘁 𝗠𝗶𝗰𝗿𝗼𝗯𝗶𝗼𝗺𝗲 𝗗𝘆𝘀𝗯𝗶𝗼𝘀𝗶𝘀

As physical therapist and clinical exercise physiologists, we are constantly looking for the "why" behind those persistent cases where a patient’s MRI doesn't match their level of pain and disability.

📘 A brand-new study by Sima et al. (2026, https://onlinelibrary.wiley.com/doi/10.1002/jsp2.70174) offers a compelling look at the "gut-disk axis" as a potential driver for chronic low back pain (LBP). Historically, we’ve relied on imaging to find structural culprits, but as Sima and Diwan (2025, https://pubmed.ncbi.nlm.nih.gov/39867670/) point out, many patients lack an easily identifiable surgical pathology. This research bridges that gap by investigating how our internal ecosystem might be fueling spinal inflammation from the inside out.

𝗧𝗵𝗲 𝗦𝗰𝗶𝗲𝗻𝗰𝗲 𝗼𝗳 𝘁𝗵𝗲 𝗚𝘂𝘁-𝗗𝗶𝘀𝗸 𝗔𝘅𝗶𝘀

The study compared 28 chronic LBP patients without advanced disk degeneration to 28 healthy, matched controls. The researchers built upon the work of Li et al. (2022, https://pubmed.ncbi.nlm.nih.gov/35286474/), who first proposed the "gut-disk axis"—a pathway where intestinal dysbiosis leads to systemic inflammation that eventually sensitizes the nerves around our spinal disks. This theory is supported by earlier findings from Shmagel et al. (2019), who found a significant association between dysbiosis and the presence and severity of musculoskeletal pain, particularly in the lower back.

📊 What Sima et al. (2026) discovered was a significant drop in alpha diversity (the richness of the microbiome) in LBP patients. This is a hallmark of dysbiosis also noted by Nitert et al. (2020, https://pmc.ncbi.nlm.nih.gov/articles/PMC7492308/) in overweight back pain cohorts. Specifically, the LBP group showed a depletion of Bacteroidota and Parabacteroides. These "good" bacteria produce short-chain fatty acids (SCFAs), which Agus et al. (2021, https://pubmed.ncbi.nlm.nih.gov/33272977/) and Ney et al. (2023, https://pubmed.ncbi.nlm.nih.gov/36977462/) have shown are vital for maintaining the gut barrier and suppressing pro-inflammatory cytokines like IL-17.

𝗪𝗵𝘆 𝗜𝘁 𝗠𝗮𝘁𝘁𝗲𝗿𝘀 𝗳𝗼𝗿 𝗢𝘂𝗿 𝗣𝗮𝘁𝗶𝗲𝗻𝘁𝘀?

When these protective bacteria are lost, "bad" players like Proteobacteria and Desulfobacterota take over. This shift increases intestinal permeability allowing endotoxins to enter the bloodstream. Once these toxins reach the intervertebral disk, they can trigger the infiltration of nociceptive fibers. This process, supported by research from Larsen (2017, https://pubmed.ncbi.nlm.nih.gov/28542929/) on Prevotella-induced inflammation and Cheng et al. (2013, https://pubmed.ncbi.nlm.nih.gov/23680281/) on Th17 cell frequency, might explain the pathogenesis and pain severity of IVD degeneration in part.

💡 Ultimately, this suggests that our role in the clinic might extend beyond biomechanics. Emerging evidence hints that therapies we already use, like exercise and psychologically informed practice, might actually help by enhancing microbial diversity. This study reinforces that we need a holistic, biopsychosocial approach to back pain that considers systemic inflammation and perhaps even microbiome-targeted interventions to get our patients moving better.

05/03/2026

Quelques exercices de stretching pour rester mobile malgré les heures de travail derrière notre bureau 😉

09/02/2026

Petit article intéressant expliquant les modalités de rupture ligamentaire du genou en ski.

C’est impressionnant et c est pourquoi le ski reste un sport qui sollicite énormément les genoux.

Les blocages au niveau de la cheville, du bassin ou du rachis peuvent augmenter les contraintes mécaniques sur les genoux adaptant les autres zones raides ou bloquées.
N’hésitez pas à venir faire un bilan avant les autres zones départ à la neige pour profiter un maximum de votre glisse ⛷️

Rdv sur Doctolib ou par téléphone
Audrey Bedouch Ostéopathe
À bientôt 👋

05/02/2026

Vous avez mal aux cervicales?

L’utilisation prolongée du téléphone portable, notamment lors de la lecture ou de la consultation des réseaux sociaux, entraîne souvent une inclinaison excessive de la tête vers l’avant.

Cette posture, adoptée de manière répétée et parfois inconsciente, sollicite fortement les muscles et les articulations de la région cervicale.

Lorsque la tête est inclinée, son poids apparent augmente considérablement pour la colonne cervicale.
Les muscles du cou doivent alors fournir un effort continu pour maintenir cette position, ce qui peut provoquer des tensions musculaires, des raideurs, des douleurs cervicales, voire des maux de tête ou des irradiations vers les épaules et le haut du dos.

Ce phénomène est parfois appelé le « text neck » ou syndrome du cou penché.

À long terme, cette mauvaise posture peut contribuer à une fatigue chronique des muscles cervicaux, à une diminution de la mobilité du cou et à une altération du confort au quotidien.

Si vous ressentez un inconfort ou des douleurs au niveau de cette région, n’hésitez pas à consulter votre ostéopathe.

À bientôt !

Audrey Bedouch Ostéopathe
Rdv Doctolib ou par téléphone
Nice Fabron - Nice Est - Fayence - Tourettes

26/01/2026

Allez un peu d’humour en cette fin de soirée 🤗🤭

31/12/2025

Je vous souhaite d’excellentes fêtes de fin d’année !!
Une belle clôture de l’année 2025 et une belle entrée dans 2026!! 🥳🥳
N’oubliez pas vos nouvelles résolutions notamment de prendre soin de vous et de votre corps!!
À bientôt ✌🏻
Audrey Bedouch ostéopathe

25/12/2025

Votre Ostéopathe vous souhaite un Joyeux Noël et de belles fêtes entourées de vos proches 🥳🎄🎁🍾

16/12/2025

Très bel article: Un petit tour du côté du nerf Vague

🧠🦠 CONCUSSION, THE VAGUS NERVE & THE BRAIN–GUT AXIS:
WHY SEROTONIN, INFLAMMATION & AUTONOMIC BALANCE MATTER MORE THAN YOU’VE BEEN TOLD

When someone suffers a concussion or head injury, the focus is almost always on the brain itself — headaches, dizziness, memory problems, visual strain, brain fog.

But neuroscience is becoming increasingly clear:

👉 The brain does not heal in isolation.
👉 The vagus nerve and the brain–gut axis play a critical role in concussion recovery.

A recent comprehensive review published in the International Journal of Molecular Sciences (MDPI) highlights how gut signaling, vagal pathways, serotonin, and neuroimmune responses directly influence brain health, inflammation, mood, cognition, and recovery after neurological injury.

⸻

🔌 The Brain–Gut Axis: A Two-Way Neurological Highway

The brain–gut axis is a bidirectional communication network connecting:

• The brain and brainstem
• The autonomic nervous system
• The immune system
• The gut microbiome
• The endocrine (hormonal) system

At the center of this network sits the vagus nerve (cranial nerve X) — the primary sensory highway sending information from the gut to the brain.

💡 Up to 80–90% of vagal fibers are afferent, meaning they carry information from the body to the brain, not the other way around.

This makes the gut one of the most powerful sensory organs influencing brain function.

⸻

🧠 What Happens to the Vagus Nerve After Concussion?

After concussion or head trauma, several things commonly occur:

🔻 Reduced vagal tone
🔻 Autonomic imbalance (sympathetic dominance / “fight-or-flight”)
🔻 Impaired heart rate variability
🔻 Increased neuroinflammation
🔻 Altered gut motility and permeability

This dysregulation can drive persistent post-concussion symptoms, including:

• Nausea and GI upset
• Anxiety and mood changes
• Poor sleep
• Fatigue
• Brain fog
• Head pressure
• Light and sound sensitivity
• Exercise intolerance

These symptoms are not psychological — they are neurophysiological.

⸻

🦠 The Gut, Inflammation & Brain Injury

The MDPI review highlights that after brain injury:

⚠️ The gut microbiome can become disrupted
⚠️ Intestinal permeability (“leaky gut”) may increase
⚠️ Immune signaling from the gut can amplify brain inflammation

This is critical because neuroinflammation delays neural recovery and interferes with synaptic plasticity — the brain’s ability to rewire and heal.

The vagus nerve normally helps suppress excessive inflammation via what’s known as the cholinergic anti-inflammatory pathway.

When vagal signaling is impaired, inflammation can remain unchecked.

⸻

🌟 SEROTONIN: THE MISSING LINK MOST PEOPLE DON’T KNOW ABOUT

One of the most important — and misunderstood — pieces of the brain-gut axis is serotonin.

🧬 Over 90% of serotonin is produced in the gut, not the brain.

Serotonin plays a critical role in:

• Mood regulation
• Sleep–wake cycles
• Pain modulation
• Cognitive flexibility
• Autonomic balance
• Neuroplasticity

Gut-derived serotonin communicates with the brain primarily through the vagus nerve.

After concussion:

🔻 Serotonin signaling can become dysregulated
🔻 Vagal feedback to brainstem nuclei is altered
🔻 Mood changes, anxiety, irritability, and depression may emerge
🔻 Sleep and circadian rhythms are disrupted

This is one reason many post-concussion patients experience emotional and psychological symptoms — even without a prior history.

Again: this is biology, not weakness.

⸻

🧠 Brainstem, Vagus & Higher Brain Centers

The vagus nerve projects directly into the nucleus tractus solitarius (NTS) in the brainstem — a key hub that connects to:

• The locus coeruleus
• The raphe nuclei (serotonin centers)
• The hypothalamus
• Limbic and emotional regulation circuits

This means vagal input from the gut can directly influence:

✔️ Arousal and alertness
✔️ Stress responses
✔️ Emotional regulation
✔️ Cognitive clarity
✔️ Recovery capacity

If this system is offline, the brain struggles to regulate itself.

⸻

🩺 Why This Matters at The Functional Neurology Center (FNC)

At FNC, we recognize that persistent concussion symptoms are often driven by network dysfunction, not structural damage alone.

That’s why our approach looks at:

🔹 Autonomic nervous system balance
🔹 Vagal tone and brainstem integration
🔹 Gut–brain signaling
🔹 Inflammatory load
🔹 Neuroplastic recovery pathways

We don’t just ask “Where does it hurt?”
We ask “Which systems are failing to communicate?”

Because restoring communication is how healing happens.

⸻

🧠✨ The Big Takeaway

Concussion is not just a brain injury.
It is a whole-system neurological event.

The vagus nerve and brain-gut axis — especially serotonin signaling — play a central role in:

• Persistent symptoms
• Mood and emotional changes
• Cognitive recovery
• Autonomic regulation
• Long-term brain health

Understanding and addressing these pathways can be the difference between stalled recovery and meaningful healing.

⸻

📩 If you or someone you love is struggling with lingering concussion symptoms, know this:

👉 There is more to the story
👉 There is a physiological explanation
👉 And there is hope

TheFNC.com
612 223 8590



https://www.mdpi.com/1422-0067/26/3/1160

Interaction of the Vagus Nerve and Serotonin in the Gut–Brain Axis
by Young Keun Hwang 1ORCID and Jae Sang Oh 1,2,