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According to the WHO, the two leading causes of death globally are cardiovascular disease and cancer — in that order.Wha...
27/08/2026

According to the WHO, the two leading causes of death globally are cardiovascular disease and cancer — in that order.

What both have in common is insulin resistance.

Early detection of insulin resistance and its prompt reversal can prevent over 70% of cardiovascular disease and cancer cases, and may reduce mortality from these conditions by more than half.

The only way to achieve this at scale is through routine insulin resistance screening: once or twice a year.

Because, by the time HbA1c, fasting blood sugar, and other standard tests begin to show abnormalities, a considerable amount of damage has already occurred.

Better still, adopting a diet and lifestyle that prevents insulin resistance from developing in the first place is the most powerful protective measure available.

Many doctors say they do not recommend fasting insulin testing because it is not in any major guideline.

But major guidelines do not determine who develops insulin resistance, or who dies from cardiovascular disease or cancer as a result.

While the guidelines catch up, take charge of your own health. Insist on a fasting insulin test.

There is no harm in running it. Only benefits.

While some major laboratories charge as much as ₦200,000, there are reputable labs that offer the test for between ₦15,000 and ₦25,000.

It is best done alongside fasting blood sugar, a fasting lipid profile, and glycated haemoglobin (HbA1c).

These enhance its interpretation.

Let us assume for a moment that medical schools around the world taught doctors everything there is to know about insuli...
26/08/2026

Let us assume for a moment that medical schools around the world taught doctors everything there is to know about insulin resistance.

It would mean that doctors understand how to identify it early. They would know which tests to use, how to interpret the results, how to connect the findings to a patient's symptoms, and how to reverse it effectively.

It would also mean they understand that insulin resistance precedes many of the most common chronic diseases — hypertension, type 2 diabetes, cancer, stroke, kidney failure, heart attack, fatty liver, and infertility — by five to fifteen years.

If that were the case, doctors would know they can detect insulin resistance long before it causes any disease, and reverse it before it does.

If they routinely encouraged patients to test for insulin resistance, they would catch it early, reverse it, and reduce the global burden of chronic disease by more than half.

Which raises an unavoidable question:

Why are they not doing it?

Why are doctors waiting for the disease to manifest before acting?

And even when the disease has manifested — why are doctors not reversing the insulin resistance that is driving it? Why is the preference, instead, to prescribe medications that suppress symptoms while the underlying condition continues to progress?

If you possess the knowledge to prevent the most lethal diseases in the world — why are you not using it?

I am genuinely asking.

26/08/2026

Yesterday, amid the noise on X, a young man reached out to me.

He had just been placed on three antihypertensives and a statin — rosuvastatin, a cholesterol-lowering drug.

When I reviewed the results on which this clinical decision was based, here is what I found:

Renal function: normal. Uric acid: normal. Lipid profile: total cholesterol 1954.5mg/dL, triglycerides 72.6mg/dL, HDL 34.8mg/dL, LDL 145.2mg/dL.

Rosuvastatin was not prescribed because his cholesterol was high. It was prescribed because his LDL was 145mg/dL.

His blood pressure was moderately elevated. His fasting blood sugar was normal — though creeping upward.

The doctor who managed this patient would probably call me a quack and insist that insulin resistance was covered in medical school.

But consider what was and was not done here.

This is a young patient with hypertension and possible dyslipidaemia. Only fasting lipid profile, euc, uric acid and fasting blood sugar were requested.

Why did this doctor not look at this patient's insulin? Why did these findings not prompt consideration of insulin resistance — the canary in the coal mine?

The answer is straightforward: insulin resistance is not taught in medical school with the depth it deserves.

When I saw the results, the first thing I assessed was the TG/HDL ratio — the surrogate marker for insulin resistance.

It indicated moderate insulin resistance. That is the root cause.

You can either proceed to address it directly or run further tests to confirm it.

Treat the root cause, not the symptoms alone.

Had I been the treating physician, I would not have prescribed rosuvastatin — and here is why: statins worsen insulin resistance.

This is an evidence-based fact.

If the root cause of a patient's problem is insulin resistance, prescribing a drug that makes it worse is not treatment.

Yet statins are routinely given to patients with type 2 diabetes, actively worsening the very condition driving their disease.

What I would have done differently:

1. Antihypertensive medication — to control blood pressure in the short term, while addressing the underlying insulin resistance. In most patients, blood pressure normalises when insulin sensitivity is restored — particularly when intervention begins early.

2. Additional investigations:
-Fasting insulin
-Glycated haemoglobin (HbA1c)
-Serum vitamin D3
-High-sensitivity CRP (hs-CRP)
-Liver function tests

3. An insulin resistance reversal protocol — structured dietary and lifestyle changes, with targeted supplementation.

Why these specific tests?

Most patients with insulin resistance have low vitamin D3 and low magnesium — which is why I check both.

hs-CRP quantifies systemic inflammation driven by insulin resistance and gives an indication of cardiovascular risk.

Many patients with insulin resistance also have fatty liver and elevated liver enzymes.

The results of these investigations would determine whether further workup is needed.

Why does all of this matter for this patient?

The treatment he has received will suppress his symptoms without addressing their cause.

In the coming years — with insulin resistance left untreated and actively worsened by statins — this young man is likely to develop type 2 diabetes.

He may also develop cancer, kidney failure, infertility, erectile dysfunction, stroke, heart attack, Alzheimer's disease, gout, or fatty liver disease.

These are the things I do and teach — for which I am called a quack.

Since I began focusing on metabolic health, I have encountered a significant number of patients with classic symptoms of...
24/08/2026

Since I began focusing on metabolic health, I have encountered a significant number of patients with classic symptoms of diabetes — yet with entirely normal fasting blood sugar and HbA1c results.

This has left them frustrated and their physicians at a loss.

Without abnormal objective markers, medications cannot be prescribed. And the standard dietary advice offered is of little use, because it is predicated on poor science.

These patients present with frequent urination — particularly at night — in the absence of any prostate pathology or urinary tract infection.

Some have the classic features of diabetic neuropathy: numbness, burning, pins and needles, and crawling sensations in the legs, hands, and scalp.

Others have diabetic microvascular complications like: erectile dysfunction and visual disturbances. Yet their fasting blood sugar and HbA1c are normal.

When I run a fasting insulin test and fasting lipid profile and assess for insulin resistance, the picture changes entirely:

Mild to severe insulin resistance, with elevated fasting insulin. It is this elevated insulin that is keeping their blood sugar and HbA1c within the normal range.

But high insulin does not protect against the symptoms and complications of diabetes. It contributes to them.

In type 2 diabetes, there are two fundamental problems: hyperglycaemia (elevated blood sugar) and hyperinsulinemia (elev...
23/08/2026

In type 2 diabetes, there are two fundamental problems: hyperglycaemia (elevated blood sugar) and hyperinsulinemia (elevated insulin).

Both are significant. Both are dangerous. In fact, a strong case can be made that hyperinsulinemia is the more dangerous of the two.

Yet no one pays attention to hyperinsulinemia. The entire clinical focus is on blood sugar, while insulin continues to cause damage in the background.

A curious observer might ask why this is the case.

The answer is straightforward: there are drugs to control blood sugar, and those drugs generate $43 billion annually in the United States alone.

There is no drug for controlling insulin, and where there is no drug, there is no revenue, and so the problem is ignored.

Meanwhile, it is hyperinsulinemia that is responsible for the complications seen in diabetic patients whose blood sugar is well controlled.

The good news is that insulin can be addressed, through a structured dietary and lifestyle plan, and nothing else.

22/08/2026

Sudden cardiac death is a real thing, and it is not caused by cholesterol. Insulin resistance, inflammation, and oxidative stress are the real culprits.

𝐖𝐡𝐲 𝐑𝐨𝐮𝐭𝐢𝐧𝐞 𝐁𝐥𝐨𝐨𝐝 𝐒𝐮𝐠𝐚𝐫 𝐓𝐞𝐬𝐭𝐬 𝐀𝐫𝐞 𝐈𝐧𝐚𝐝𝐞𝐪𝐮𝐚𝐭𝐞⁣​⁣Fasting and random blood sugar tests are like a faulty smoke detector — o...
22/08/2026

𝐖𝐡𝐲 𝐑𝐨𝐮𝐭𝐢𝐧𝐞 𝐁𝐥𝐨𝐨𝐝 𝐒𝐮𝐠𝐚𝐫 𝐓𝐞𝐬𝐭𝐬 𝐀𝐫𝐞 𝐈𝐧𝐚𝐝𝐞𝐪𝐮𝐚𝐭𝐞⁣
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Fasting and random blood sugar tests are like a faulty smoke detector — one that only sounds the alarm after the fire has already destroyed the building.
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They act far too late. Here is why. ⁣
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Blood sugar is tightly regulated because even a slight sustained rise damages major structures in the body. ⁣
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The body will do almost anything to keep it within the normal range, and insulin is the primary mechanism by which it does so.
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Whenever blood sugar rises, the pancreas secretes insulin to facilitate the movement of glucose from the bloodstream into cells, restoring normal levels. ⁣
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This is normal physiology.
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The problem arises when we repeatedly do things that raise blood sugar above normal. ⁣
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The pancreas responds by producing progressively more insulin to keep blood sugar in check. ⁣
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Blood sugar remains normal, but insulin is rising. ⁣
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Once insulin levels exceed approximately 6 to 7 µIU/mL, it begins causing its own damage – independent of blood sugar. ⁣
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The conditions driven by chronically elevated insulin include cancer, hypertension, chronic kidney disease, neuropathy, obesity, PCOS/PMOS, infertility, erectile dysfunction, and fatty liver disease, among others. ⁣
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All of this occurs while blood sugar remains perfectly normal.
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Insulin continues to rise to compensate — until, eventually, the pancreas becomes exhausted and can no longer meet the body's demands. ⁣
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Insulin secretion plateaus or falls. Blood sugar begins to climb. ⁣
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The fasting blood sugar alarm finally sounds.
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This is the moment your doctor tells you there is a problem.
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It takes five to fifteen years to reach this point. ⁣
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During those years, every condition driven by elevated insulin has been developing unhindered. ⁣
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This is why most people, at the time of their diabetes diagnosis, already have complications — hypertension, arthritis, infertility, erectile dysfunction, recurrent infections, or cancer.
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When the clinical focus shifts from blood sugar to insulin, diabetes and other chronic diseases can be detected before they even begin.
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This is precisely why this topic is not taught in any medical school anywhere in the world. ⁣
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If doctors understood and applied this knowledge, the incidence of diabetes, kidney failure, hypertension, cancer, and most other chronic diseases would fall by more than half. ⁣
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And if this happens, who would then buy the drugs?
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Your guess is as good as mine…⁣
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You can choose to remain within a system that is designed to continually profit from your illness, or you can take ownership of your health and pursue a longer, healthier life.
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Whatever your choice, always remember that…
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Fasting and random blood sugar tests are faulty smoke detectors. A fasting insulin test is one that actually works.

21/08/2026

We are failing to pick chronic kidney disease and kidney failure early because we act only when fasting blood sugar becomes abnormal.

Kidney damage begins years before blood sugar testing reveals anything unusual. By the time the blood sugar test flags a problem, significant damage may already have been done.

If we continue to wait for that threshold to be crossed, the trajectory of kidney failure will not change.

Fasting insulin testing would allow us to detect and prevent kidney damage before it even begins.

We can't continue to speak about prevention while ignoring this….

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