What are the opening hours?

Monday 9am - 5pm Tuesday 9am - 5pm Wednesday 9am - 5pm Thursday 9am - 5pm Friday 9am - 5pm

http://www.mycareclinicatlanta.com/

MyCare Clinic, 3941 Holcomb Bridge Road Suite 100, Peachtree Corners, GA (2026)

10/12/2026

FASCINATING! To prevent diabetes, you don't actually have to change what you eat tonight. Just the order in which you do so.

Researchers at Kyoto University published a study this week in the journal Diabetes showing that taking a small amount of olive oil before eating carbohydrates, rather than during or after the meal, significantly reduced the early blood sugar spike that follows a carb-heavy meal. Even in mice with type 2 diabetes and severe insulin deficiency, olive oil taken first still blunted the blood sugar rise. The mechanism turned out to be more interesting than anyone expected, and the practical takeaway takes about 30 seconds to act on.

What the science actually shows

When olive oil reached the gut before carbohydrates, it triggered an early release of two hormones, glucagon and GLP-1, working together with the vagal nerve to slow gastric emptying. In plain terms: the stomach held onto its contents longer, which meant glucose entered the bloodstream more gradually rather than flooding in all at once. Mice that got oil first had significantly lower blood sugar 15 and 30 minutes after eating compared to those who got oil during or after the meal. The timing was the entire variable. Same food, same amount, completely different metabolic response based on what came first. The researchers specifically noted that this builds on existing clinical evidence suggesting that eating vegetables, protein, or fat before carbohydrates produces smaller blood sugar spikes, and that their study now provides a clear biological explanation for why the order matters.

Why blood sugar spikes matter for everyone, not just diabetics

Most people think about blood sugar as a diabetes concern. It isn't, or at least it shouldn't only be. Post-meal blood sugar spikes affect energy, hunger, mood, inflammatory markers, and over time contribute to insulin resistance that sets the stage for metabolic disease, weight gain, and cardiovascular risk years before a diabetes diagnosis ever comes. The afternoon energy crash most people experience after a carb-heavy lunch is largely a blood sugar spike and subsequent drop playing out in real time. Managing those spikes through how and what you eat is one of the most accessible metabolic health tools available, and this research adds a specific, practical piece to that picture. A tablespoon of olive oil or a handful of nuts before your bread, pasta, or rice changes what that meal does to your blood sugar in a measurable way.

What to actually do with this

The olive oil timing finding adds to a body of evidence around meal sequencing that is genuinely useful and costs nothing to implement. Eat your salad, vegetables, or a small amount of healthy fat first. Then your protein. Then your carbohydrates. A drizzle of olive oil on vegetables before the pasta arrives, a few bites of avocado before the rice, a small handful of almonds before the sandwich. None of this requires overhauling your diet, buying a supplement, or tracking anything. It is a sequencing strategy that takes seconds to implement and has a measurable effect on your metabolic response to every meal you eat for the rest of your life. For anyone on a GLP-1 medication, it works with the same hormonal pathway the medication targets, which means it amplifies rather than duplicates what the medication is already doing.

One small habit. Every meal. Every day. That compounds into something significant.

Rasa Teytel, FNP-C at MyCare Clinic works with patients on the practical nutritional strategies that support weight management and metabolic health alongside medical treatment. If blood sugar, insulin resistance, or weight management is part of your health picture, this kind of conversation is exactly what a consultation is for. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: If you are on a GLP-1 medication and still experiencing energy dips or hunger between doses, meal sequencing and the composition of what you eat before carbohydrates can make a meaningful difference to how steady your blood sugar stays through the day. Worth trying before adjusting your dose.

Are you going to try this tonight? Drop a comment, and tag someone who eats the bread first at every restaurant and needs to know there is a better way.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

10/09/2026

Another miraculous discovery for those suffering from chronic pain! For the first time in the history of medicine, scientists have figured out how to make cartilage grow back. If you have been told your knee pain is just something you have to live with, this is the week that changes that conversation.

Stanford Medicine published a landmark study today in Science, one of the most respected research journals in the world, showing that blocking a single aging-related protein caused worn knee cartilage to regrow in older mice, restored naturally lost cartilage after ACL-like injuries, and here is the part that stopped the research community in its tracks: human cartilage samples collected during knee replacement surgeries responded to the same treatment by beginning to produce new, functional cartilage. Not in mice. In human tissue. Today.

What they found and why it matters so much

The protein at the center of this discovery is called 15-PGDH. As the body ages, this protein accumulates in joint tissue and essentially acts as an off switch for cartilage regeneration. The more of it your joints have, the less your body can repair the cartilage that daily movement, aging, and injury gradually wear away. The Stanford researchers identified a small molecule inhibitor that blocks 15-PGDH and in doing so releases the brake on cartilage regeneration. In older mice whose knee cartilage had naturally thinned with age, the treatment caused cartilage to thicken across the entire joint surface. In mice with ACL-like injuries, it prevented the development of osteoarthritis entirely. The lead researcher described the regeneration as dramatic and beyond anything previously reported with any other intervention. Those are not words scientists use casually.

What it means for the 32 million Americans living with osteoarthritis right now

Osteoarthritis is the most common form of arthritis and one of the leading causes of chronic pain and disability in adults worldwide. The fundamental problem with every existing treatment is that none of them actually repair the damage. Cortisone injections manage inflammation. Hyaluronic acid injections lubricate the joint. Physical therapy builds the muscle around the damage. Joint replacement surgeries remove the affected joint entirely and install a mechanical substitute. Every single one of these approaches works around the cartilage problem. None of them solve it. What Stanford has identified is the first biological pathway that could potentially reverse cartilage loss rather than compensate for it. That is a category change in how arthritis treatment is thought about, and it matters for everyone whose knees, hips, and spine have been paying the price of decades of wear.

What to be realistic about and what to do in the meantime

This is a mouse study with human tissue results that are promising but preliminary. Clinical trials in humans are the necessary next step and they take time. The treatment is not available today and will not be available in a clinic next year. What today's research does is open a door that the medical community has been trying to find for decades, and it confirms that the biological machinery for cartilage regeneration exists in human tissue and can potentially be activated. That is not nothing. That is the foundation of what becomes a treatment. In the meantime, the interventions that protect remaining cartilage, manage inflammation, and preserve joint function are not placeholders. They are what determines what condition your joints are in when this research becomes a clinical reality, and that timeline matters enormously for how much cartilage you still have to work with.

Your joints are worth protecting right now, not just managing.

Rasa Teytel, FNP-C at MyCare Clinic offers image-guided procedures that target joint pain with precision, including knee injections, soft tissue treatments, and diagnostic evaluations that identify exactly what is driving your pain and what can be done about it today. If you have been told to just manage your knee pain, come in for a second opinion on what managing it well actually looks like. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: Cartilage loss is irreversible with current treatments, which means protecting what you have now matters more than most patients are told. Anti-inflammatory diet, targeted strengthening, appropriate weight management, and precision joint injections that reduce destructive inflammation are all tools that slow the process. Come in before the damage makes the conversation harder.

Did you see this study today? Drop a comment, and tag someone who has been living with knee pain and waiting for better options. Better options are coming.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

10/05/2026

Cut your high LDLcholesterol in half with a single treatment and not think about it again for a year? That just happened. In real patients. In a peer-reviewed trial.

Researchers at the Cleveland Clinic published results this week showing that a one-time infusion of an experimental gene-editing treatment called CTX310 reduced LDL cholesterol by 53 percent and triglycerides by 48 percent in patients, with those results holding steady for a full year after a single dose. The results were published in the New England Journal of Medicine, one of the most respected medical journals in the world, and presented at the European Society of Cardiology's annual meeting this past August. This is genuinely new territory in medicine, and it is worth understanding what it means and what it doesn't.

What CRISPR actually did here, in plain language

Your liver produces a protein called ANGPTL3. Think of it as a brake system that slows your body's ability to clear cholesterol from the bloodstream. CTX310 uses CRISPR gene-editing technology to find that specific gene in your liver cells and switch it off permanently. Once the gene is edited, the brake releases and your body clears cholesterol more efficiently on its own, without a daily pill, without a monthly injection, without ongoing medication of any kind. LDL cholesterol levels began dropping within two weeks of the infusion and stayed low for the entire year of follow-up. The researchers were expecting a 30 to 40 percent reduction to count as a success. They got 53 percent. The lead cardiologist from Cleveland Clinic called it a very big deal. He is not wrong.

What this means for the millions of Americans managing high cholesterol

High LDL cholesterol is one of the primary drivers of the arterial plaque buildup that causes heart attacks and strokes. It is the same process that likely contributed to the sudden death of Senator Lindsey Graham earlier this year, and it is currently managed in most patients through daily statin medications that require lifelong compliance and don't work equally well for everyone. Statins are genuinely effective for many people but come with side effects that cause a significant number of patients to stop taking them. The promise of a one-time treatment that does the same job more dramatically and without daily compliance is not a minor development. It is potentially a fundamental shift in how cardiovascular disease gets managed. OHSU enrolled its first West Coast participant in the expanded trial just two days ago, which means the research is actively moving forward right now.

What to be clear-eyed about before getting too excited

This is a phase 1 trial. Fifteen people. No placebo group. Followed for one year. Those are small numbers by the standards that lead to FDA approval, and the researchers are appropriately careful about what they can claim. The trial did not measure heart attacks or strokes because no study this size could do that meaningfully. Longer-term safety follow-up is planned but not yet complete. Gene editing in the liver is a significant intervention and the full picture of what it does over a decade is not yet known. What this study proves is safety in the short term and impressive efficacy at one year. What it opens is a research pathway that, if larger trials confirm these results, could eventually give millions of people with stubborn high cholesterol a treatment option that doesn't exist today.

The future of cardiovascular medicine is being written right now. This is one of the most significant chapters it has produced in years.

In the meantime, the tools that exist today are powerful and underused. Rasa Teytel, FNP-C at MyCare Clinic offers comprehensive cardiovascular and metabolic evaluations including full lipid panels, inflammatory markers, and personalized prevention planning. If you have not had your cholesterol checked recently, or if you have been managing high LDL without a clear plan, come in before the end of the year while your insurance benefits are still in full effect. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: LDL cholesterol and triglycerides are only part of the cardiovascular picture. Apolipoprotein B, C-reactive protein, and fasting insulin together tell a far more complete story about your actual heart disease risk than a standard lipid panel alone. Ask about a full cardiovascular workup at your next visit.

Had you heard about this? Drop a comment, and share this with someone who has been managing high cholesterol for years and could use some genuinely good news.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

10/01/2026

Two women. That is how many will be diagnosed with breast cancer in Georgia today. And tomorrow. And every day for the rest of this year.

This October, 10,440 Georgia women will receive a breast cancer diagnosis. 1,430 will die from it. Nationally, 321,910 new cases. 42,140 deaths. Behind every one of those numbers is a name, a family, a life interrupted or ended by a disease that in many cases was entirely survivable if it had been caught sooner. That last part is not a consolation. It is the whole point of this post.

The survival numbers are staggering and almost nobody talks about them plainly enough

When breast cancer is detected early, before it has spread beyond the breast, the five-year survival rate is over 99 percent. Ninety-nine percent. When it has spread to distant parts of the body, that number drops to 28 percent. The difference between those two outcomes is not treatment. It is not genetics. It is not luck. In most cases, it is timing. It is whether a mammogram was scheduled and kept, or delayed another year because life got busy, because it felt like an overreaction, because it was uncomfortable, because there was no family history and it seemed unnecessary. Breast cancer does not wait for a convenient time. Early detection is the single most powerful intervention in breast cancer medicine, and it is available to every woman reading this post right now.

The women most at risk are often the least likely to screen

The statistics reveal a painful disparity that October demands we name directly. Black women in Georgia and across the United States are diagnosed with breast cancer at similar rates to white women but die from it at significantly higher rates. The gap is not biological. It is driven by delayed access to screening, later-stage diagnoses, gaps in insurance coverage, and barriers in navigating a healthcare system that has not always shown up equally. It is also driven by the cultural silence around cancer that keeps women from talking about symptoms, from pushing for referrals, from insisting on follow-up when something feels wrong. Awareness without access changes nothing. This month, if you know a woman in your community who has not been screened, this post is worth more than a share. It is worth a phone call.

What to do this week, not this month

Breast cancer awareness month produces a lot of pink. What it needs to produce is appointments. A mammogram can detect breast cancer before a lump can be felt or any symptom appears, and for many women that window of invisible, early-stage disease is the only window that results in the outcome they deserve. Current guidelines recommend annual mammograms beginning at 40 for average-risk women, and earlier for those with a family history, dense breast tissue, or BRCA gene mutations. If you do not know your risk profile, that is the first conversation to have. If you know your risk and have been putting off a screening, this is the week. Not the end of October. This week.

Breast cancer death rates have dropped 44 percent since 1989. That progress was built on screening. It was built on women showing up for themselves before they had a reason to be afraid. It was built on the appointments that got made.

At MyCare Clinic, Rasa Teytel, FNP-C can assess your breast cancer risk, coordinate mammogram referrals, and make sure you have the clinical support you need to act on what screening finds. If you are overdue, come in this week. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: Dense breast tissue, which affects about 40 percent of women, can make standard mammograms less reliable at detecting early tumors. If you have been told you have dense breasts, ask your provider specifically about supplemental screening options. It is a conversation worth having before you need it.

Share this post with every woman in your life who has been putting off her mammogram. Not as a reminder. As an ask. The appointment you help someone make this October may be the one that matters most.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

09/28/2026

Newest Research: Results of 615 Adult MRI Scans

Your knee pain might not be coming from your knee.

Researchers just published a study in Radiology, one of the top imaging journals in medicine, that found something worth paying attention to. MRI scans of 615 adults showed that people who ate more ultra-processed foods had significantly more fat built up inside their thigh muscles, the exact muscles responsible for holding the knee stable and absorbing impact with every step. Not belly fat. Not fat under the skin. Fat working its way inside the muscle tissue itself, showing up on scan as streaks of fatty degeneration where healthy muscle fiber should be. The part that surprised even the researchers: it didn't matter how many calories people were eating, how active they were, or what they weighed. The association held anyway. What people ate, not just how much, was driving what their muscles looked like on the inside.

Why fat inside your thigh muscles is a knee problem

Your thigh muscles are the knee's primary support system. They absorb load, stabilize the joint, and protect the cartilage from taking direct impact with every movement. When fat infiltrates that muscle tissue, the muscle gets weaker and less responsive. It stops doing its job as well. And when thigh muscles stop doing their job, the knee starts absorbing more of what the muscle was supposed to handle. That is a recipe for the kind of wear and tear that becomes osteoarthritis. Previous research had already connected declining thigh muscle quality to knee osteoarthritis progression. This study added something new: a specific dietary driver that has never been this clearly identified before.

The number that puts this in perspective

The average study participant was getting 41 percent of their daily food from ultra-processed sources. That is not an outlier. In the United States, ultra-processed foods make up more than half of what most adults eat on any given day. Packaged snacks, deli meats, frozen meals, sugary cereals, fast food, sodas, flavored protein bars. These are not occasional indulgences for most people. They are the backbone of how America eats. And MRI is now showing us what that looks like inside the muscles that hold our bodies upright. The damage is invisible in its early stages. You cannot feel intramuscular fat building up. You can only feel what it eventually produces, and by the time it produces something, the process has usually been running for years.

What to do with this right now

The researchers are clear that this study shows an association, not a proven cause and effect. Longer studies tracking the same people over time are still needed. But the signal is strong, consistent across the full dataset, and independent of the factors you would normally expect to explain it. The practical takeaway is not complicated. Swap out ultra-processed staples for whole food alternatives where you can, build lower body strength through regular resistance training, and if your knees are already giving you trouble, get a proper evaluation now rather than waiting for the problem to get louder. Muscle fat infiltration visible on MRI is not where you want to start the conversation. It is what a good clinical plan is designed to prevent.

Your diet is not just showing up in your bloodwork and on your scale. It is showing up in the muscles that hold your joints together. That is a connection worth taking seriously before it shows up as pain you cannot ignore.

Rasa Teytel, FNP-C at MyCare Clinic evaluates and treats knee pain using image-guided procedures that target the actual source of the problem, not just the symptoms around it. If your knees have been bothering you and you have been putting off finding out why, this is a good week to come in. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: Knee pain without a clear injury behind it, or pain that keeps coming back despite rest and medication, often has an inflammatory or structural driver that a proper evaluation can identify. Your diet, your inflammatory markers, and the condition of the supporting muscles around your knee all tell a more complete story than an X-ray alone.

Did this one surprise you? Drop a comment, and tag someone whose knees have been hurting and who hasn't thought twice about what's in their grocery cart.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

09/25/2026

Fall Season Warning: When did you last have your liver health evaluated?

Your liver is quietly one of the hardest working organs in your body. It is also the one most people never think about until something goes seriously wrong.

As Atlanta moves into fall and the eating patterns that come with it, cooler weather, heavier meals, more social eating, more alcohol at gatherings, the conditions that drive fatty liver disease get quietly worse for millions of people who have no idea the process is already underway. One in three American adults has fat accumulating in their liver right now. Most of them feel completely fine. That is precisely what makes this the most underdiagnosed chronic condition in the country, and precisely why fall is the right time to pay attention.

What is actually happening in a fatty liver

Fatty liver disease, now clinically called metabolic dysfunction-associated steatotic liver disease, develops when excess fat accumulates in liver cells faster than the liver can process it. The drivers are familiar: high calorie intake, refined carbohydrates, sugar, alcohol, insulin resistance, and a sedentary lifestyle. What makes it different from other metabolic conditions is how quietly it progresses. Early-stage fatty liver causes no pain, no visible symptoms, and no reliable signals in standard bloodwork until the disease has already advanced. Lund University researchers publishing in 2026 identified specific genetic and metabolic markers that shift as fatty liver progresses toward fibrosis, the stage where scar tissue begins to replace healthy liver cells. Once fibrosis becomes cirrhosis, the damage is largely irreversible. The entire trajectory from early fat accumulation to serious disease can span a decade of complete silence.

The fall-specific risk most people overlook

Summer eating tends to be lighter. Produce is abundant, appetites are smaller in the heat, and people move more. Fall reverses most of that. Heavier, higher-calorie comfort meals replace salads and grilled fish. Alcohol consumption rises through the holiday season. Activity levels drop as temperatures fall and schedules tighten. For a liver managing early-stage fat accumulation, that seasonal shift in diet and lifestyle is genuinely significant. Research published in 2026 in Nature Communications identified distinct metabolic subtypes of fatty liver disease with different inflammatory and fibrotic profiles, suggesting that dietary patterns influence not just whether fatty liver develops but how aggressively it progresses. Fall is not a neutral season for liver health. For most people it is the most liver-stressful time of year, running from October through January, and it starts now.

What the latest research says actually helps

A vitamin B breakthrough published earlier this year showed meaningful reductions in liver fat accumulation in research models, adding to a growing body of evidence pointing toward specific nutritional interventions that directly influence liver metabolism. Daily tomatoes, as covered in a post earlier this month, showed measurable reductions in liver fat in a six-week randomized trial. Intermittent fasting on a 5:2 schedule has been shown to protect against liver inflammation and reduce the risk of progression toward liver cancer in fatty liver patients. Semaglutide, the active ingredient in Ozempic and Wegovy, is currently in the ESSENCE trial showing significant reductions in metabolic liver disease without worsening scarring. And the gut microbiome research published in 2026 confirms that the balance of bacteria in your intestine directly influences whether your liver accumulates fat or resists it. None of these are distant future therapies. They are available, evidence-backed interventions that can be built into a clinical plan right now.

The most useful thing about fatty liver disease is that it is highly reversible in its early stages. The least useful thing is that most people never find out they have it until it isn't early anymore.

At MyCare Clinic, Rasa Teytel, FNP-C offers in-house Velacur liver assessments and elastography, non-invasive scans that evaluate liver fat and stiffness without a referral, without imaging center appointments, and without waiting weeks for answers. If findings are clinically significant, they can also open the door to GLP-1 therapy that may be covered by your insurance. Before the holiday season gets fully underway and the eating patterns of fall take hold, this is the right time to know where your liver actually stands. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: Mildly elevated liver enzymes on routine bloodwork are one of the few early signals fatty liver disease produces, and they are easy to dismiss as a minor finding. If your ALT or AST has been creeping up over the past year or two, that trend is worth evaluating with a proper liver assessment before it tells a louder story.

When did you last have your liver health evaluated? Drop a comment, and share this with someone heading into fall who could use a reason to check in before the holidays arrive.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

09/22/2026

Early Alzheimer's Prevention: By the time most people get an Alzheimer's diagnosis, the disease has been developing in their brain for 15 to 20 years. That window is about to change.

Sunday is World Alzheimer's Day, and this year it arrives with some of the most significant research the field has ever produced. At the Alzheimer's Association International Conference in London this past July, scientists presented findings that shift the entire conversation around when Alzheimer's can be detected, how early intervention can begin, and what any of us can do right now to protect our brain health. Here is what came out of that conference that every adult should know.

A simple blood test may detect Alzheimer's a decade before symptoms appear

For most of Alzheimer's history, meaningful diagnosis required expensive brain imaging, spinal taps, or waiting for symptoms that appear only after significant damage has already occurred. That is changing fast. Researchers at AAIC 2026 found that a blood test measuring a protein called p-tau217, a hallmark marker of Alzheimer's disease, could predict cognitive decline up to ten years before any memory or thinking problems appear in some patients. A separate study found that primary care physicians using these blood test results diagnosed Alzheimer's with nearly the same accuracy as dementia specialists. Ten years of lead time. At your family doctor's office. That is not a minor development. It is a fundamental shift in what early detection means for this disease, and it means the conversation about brain health belongs in your regular checkup, not just a neurologist's office.

Lifestyle intervention is now clinically proven to slow cognitive decline

This is the finding most directly relevant to what any of us can do starting this week. A large-scale lifestyle intervention study conducted across 11 countries in Latin America showed that a structured program combining physical exercise, nutrition, cognitive training, and social engagement measurably improved memory and cognitive function in older adults at high risk for dementia. This builds on similar US findings and extends them to diverse populations across different backgrounds and socioeconomic levels. The conclusion the researchers drew was direct: brain health can be improved across a wide range of people through lifestyle, and the earlier those changes begin, the more protective they are. Exercise, anti-inflammatory diet, sleep, social connection, and mental engagement are not soft wellness advice. They are clinically validated interventions for one of the most feared diseases of our time.

The drug pipeline is the most promising it has ever been

For anyone following Alzheimer's research with a family member in mind, this context matters. Researchers are currently evaluating 158 medicines across 192 active trials, a 40 percent increase over the past decade. Critically, the field is moving beyond its long focus on amyloid, the protein that became the dominant research target for years with limited clinical results. Treatments targeting tau, reducing neuroinflammation, and rebalancing the immune system have risen sharply in the pipeline. Injectable versions of existing treatments are in development to improve accessibility and cost. The science is not standing still, and neither is the hope.

More than 7 million Americans are currently living with Alzheimer's, a number projected to nearly double by 2060. Most of them received their diagnosis years after the disease began. The research presented this year is building a future where that gap closes significantly, where a blood test at a primary care visit catches a risk that would have been invisible until symptoms appeared. We are not there yet everywhere, but the direction is clear and the pace is accelerating.

Rasa Teytel, FNP-C at MyCare Clinic takes brain health seriously as part of every whole-body evaluation, looking at metabolic function, inflammation, hormonal health, and sleep, all of which have documented connections to cognitive decline and dementia risk. If brain health is a concern for you or someone in your family, that conversation starts here. Appointments preferred, walk-ins always welcome.

Visit www.mycareclinicatlanta.com

MyCare Clinic Pro Tip: Insulin resistance, chronic inflammation, poor sleep, and thyroid dysfunction are all measurable factors that research now links to accelerated cognitive aging. If you have not had a full metabolic workup recently, you may be missing part of your brain health picture without knowing it.

Do you have a family member affected by Alzheimer's? Share this post on World Alzheimer's Day, and tag someone who should know that the science is moving faster than most people realize.

MyCare Clinic - Rasa Teytel, FNP-C
Visit Us: 3941 Holcomb Bridge Rd Suite 100, Peachtree Corners, GA 30092
Call Us: 678-500-8985
Email Us: [email protected]

MyCare Clinic

10/12/2026

10/09/2026

10/05/2026

10/01/2026

09/28/2026

09/25/2026

09/22/2026

Address

Opening Hours

Telephone

Website

Alerts

Contact The Business

Shortcuts

Share

Category

Listing takedown request

Monday	9am - 5pm
Tuesday	9am - 5pm
Wednesday	9am - 5pm
Thursday	9am - 5pm
Friday	9am - 5pm